A mechanism of resistance to HIV-1 entry: Inefficient interactions of CXCR4 with CD4 and gp120 in macrophages

Dimiter S. Dimitrov*, David Norwood, Tzanko S. Stantchev, Yanru Feng, Xiaodong Xiao, Christopher C. Broder

*Corresponding author for this work

Research output: Contribution to journalArticlepeer-review

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Abstract

To test the hypothesis that inefficient interactions of CXCR4 with CD4 and gp120 could affect HIV-1 entry, we incubated macrophages, monocytes, and lymphocytes with gp120 and coimmunoprecipitated CD4 by using anti-CXCR4 antibodies. CD4 was efficiently coimmunoprecipitated in lymphocytes and monocytes but not in macrophages. Overexpression of CD4 in macrophages resulted in detection of CD4-CXCR4 and gp120-CD4-CXCR4 complexes in parallel with the restoration of macrophage fusion susceptibility. These results suggest a mechanism of resistance to entry of some X4 HIV-1 strains into macrophages and a method for dissection of the initial stages of HIV entry.

Original languageEnglish
Pages (from-to)1-6
Number of pages6
JournalVirology
Volume259
Issue number1
DOIs
StatePublished - 20 Jun 1999

Keywords

  • CD4
  • Chemokine receptors
  • Coreceptors
  • CXCR4
  • Envelope glycoprotein
  • HIV-1
  • Membrane fusion

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