Sudden infant death syndrome: Gene-environment interactions

Carl E. Hunt; Fern R. Hauck

doi:10.1007/978-1-84882-927-5_2

Sudden infant death syndrome: Gene-environment interactions

Carl E. Hunt^*
, Fern R. Hauck

^*Corresponding author for this work

Pediatrics

Research output: Chapter in Book/Report/Conference proceeding › Chapter › peer-review

3 Scopus citations

Abstract

Sequencing of the human genome has resulted in a rapidly expanding understanding of the molecular basis of many human diseases and the incredible complexity of genotype-phenotype relationships.^1-5 Some genes are expressed only in healthy individuals or in disease conditions, only at specified ages, or in response to specific perturbations or states (e.g., sleep). Some genes, thus contribute to susceptibility to disease, but other genes and their polymorphisms contribute to protection against illness.

Original language	English
Title of host publication	Clinical Approach to Sudden Cardiac Death Syndromes
Publisher	Springer London
Pages	7-21
Number of pages	15
ISBN (Print)	9781848829268
DOIs	https://doi.org/10.1007/978-1-84882-927-5_2
State	Published - 2010

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title = "Sudden infant death syndrome: Gene-environment interactions",

abstract = "Sequencing of the human genome has resulted in a rapidly expanding understanding of the molecular basis of many human diseases and the incredible complexity of genotype-phenotype relationships.1-5 Some genes are expressed only in healthy individuals or in disease conditions, only at specified ages, or in response to specific perturbations or states (e.g., sleep). Some genes, thus contribute to susceptibility to disease, but other genes and their polymorphisms contribute to protection against illness.",

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AU - Hauck, Fern R.

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N2 - Sequencing of the human genome has resulted in a rapidly expanding understanding of the molecular basis of many human diseases and the incredible complexity of genotype-phenotype relationships.1-5 Some genes are expressed only in healthy individuals or in disease conditions, only at specified ages, or in response to specific perturbations or states (e.g., sleep). Some genes, thus contribute to susceptibility to disease, but other genes and their polymorphisms contribute to protection against illness.

AB - Sequencing of the human genome has resulted in a rapidly expanding understanding of the molecular basis of many human diseases and the incredible complexity of genotype-phenotype relationships.1-5 Some genes are expressed only in healthy individuals or in disease conditions, only at specified ages, or in response to specific perturbations or states (e.g., sleep). Some genes, thus contribute to susceptibility to disease, but other genes and their polymorphisms contribute to protection against illness.

UR - https://www.scopus.com/pages/publications/84877033617

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DO - 10.1007/978-1-84882-927-5_2

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SN - 9781848829268

SP - 7

EP - 21

BT - Clinical Approach to Sudden Cardiac Death Syndromes

PB - Springer London

ER -

Sudden infant death syndrome: Gene-environment interactions

Abstract

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